Common Causes of Hair Loss in Women

Hair loss in women is far more common than most realize, affecting over 50% of women at some point in their lives. Yet, it remains a taboo topic for many. Understanding the root causes of hair loss in women is the first step to finding lasting solutions. While age and genetics play a role, many women experience excessive hair loss due to hormonal fluctuations, stress, autoimmune conditions, and underlying health issues.

Some of the most common causes of hair loss in women are: 

• Female Pattern Hair Loss (Androgenetic Alopecia)
• PCOS-Related Hair Loss
• Thyroid Hair Loss (Hypothyroidism & Hashimoto’s)
• Stress-Induced Hair Loss (Telogen Effluvium)
• Autoimmune Hair Loss (Alopecia Areata & Lupus)
• Postpartum Hair Loss (Postpartum Alopecia)

One emerging focus in the science of hair health is a protein called FGF5. This protein regulates the hair growth cycle by signaling the end of the growth phase, leading to shedding. When FGF5 is overactive, hair may prematurely enter the shedding phase. Targeting this protein with FGF5 blockers has shown promise in extending the growth phase and improving hair density.

évolis®, a leader in hair science, formulates products that support hair wellness at the root. Our range is backed by research and designed to tackle common causes of hair loss in women. In this blog, we’ll explore how hormones, stress, autoimmunity, and lifestyle factors contribute to female hair loss and how you can take control of your hair health.

Understanding the Hair Growth Cycle

The Phases of Hair Growth

Hair growth occurs in a cyclical processfour main stages

• Anagen (Growth Phase): This is the active phase when hair grows from the follicle. It can last 2-7 years.
• Catagen (Transition Phase): A brief 2-3 week phase where the follicle begins to shrink.
• Telogen (Resting Phase): Lasting around 3 months, the hair stops growing but remains anchored.
• Exogen (Shedding Phase): The hair detaches and falls out, making room for a new strand.

How Hair Loss Occurs

Hair loss occurs when this cycle is disrupted. The most common causes include:

• Hormonal Imbalances: Shifts in estrogen, progesterone, or androgens can prematurely push hair into the telogen phase.
• Stress: Chronic stress increases cortisol, which interferes with follicle activity.
• Autoimmune Disorders: The immune system may mistakenly attack hair follicles, triggering shedding.
• Aging: Natural aging shortens the anagen phase and reduces follicle productivity.
• Understanding these triggers allows for targeted interventions that can restore balance to the hair cycle.

 

Female Pattern Hair Loss (Androgenetic Alopecia in Women)

Androgenetic alopecia is the most common form of hair loss in women. It’s a genetically influenced condition primarily driven by the hormone dihydrotestosterone (DHT), a more potent derivative of testosterone. While testosterone and DHT are commonly associated with male physiology, women also produce small amounts of these androgens in the adrenal glands and ovaries.

1. Conversion of Testosterone to DHT

In the body, the enzyme 5-alpha-reductase converts testosterone into DHT. There are two main types of this enzyme—Type I and Type II—with Type II being most active in the hair follicles and scalp.

In women with a genetic predisposition, the hair follicles have an increased number of androgen receptors or a higher sensitivity to DHT.

2. DHT Binds to Hair Follicle Receptors

DHT binds to these androgen receptors at the base of the hair follicle—specifically within the dermal papilla, which controls the activity of the follicle and plays a major role in regulating hair growth.

3. Follicle Miniaturization Begins

Over time, repeated exposure to DHT triggers a process called follicle miniaturization:

• The anagen(growth)phase of the hair cycle becomes shorter.
• The telogen(resting) phase is prolonged.
• Hair strands become finer, shorter, and less pigmented with each cycle.
• Eventually, the follicle becomes so small that it produces vellus-like hairs—thin, wispy strands that offer little to no coverage.

This process is not due to follicle death but a progressive weakening of its function.

4. Why Miniaturization Leads to Shedding

As the growth cycle shortens, hairs enter the telogen phase more frequently and for longer periods. Because fewer hairs are in the active growth (anagen) phase at any given time, this results in overall thinning—especially on the crown, temples, or part line.

Additionally, these weakened follicles become more sensitive to inflammatory signals and oxidative stress, which can further hair loss.

In later stages, some follicles may go dormant entirely, leading to more visible scalp exposure.

Why Women Experience Thinning, Not Balding

Unlike men—who often develop receding hairlines or bald spots—women typically experience diffuse thinning across the crown or part line, while the frontal hairline is usually preserved. This is because:

• Women have lower overall androgen levels.
• Estrogen, which has protective effects on hair, can counterbalance the impact of DHT to some extent.
• The distribution and sensitivity of androgen receptors differ between the sexes.

Supporting the Hair Follicles 

Alongside DHT, a protein called FGF5 (Fibroblast Growth Factor 5) also plays a role in signaling the hair follicle to transition from the growth phase to the resting phase. In those experiencing female pattern hair loss, both DHT and FGF5 can work in tandem to shorten the anagen phase, accelerating the cycle of thinning and shedding.

Blocking DHT with medical or natural therapies and using FGF5 inhibitors, like those found in évolis, may slow progression and encourage regrowth. Medical consultation is advised for diagnosis and treatment plans. Blocking FGF5, as seen in some botanicals used in évolis formulations, may help to prolong the anagen phase, counteract miniaturization, and encourage the return of healthier, denser growth while you work with your physician to treat the symptoms associated with overactive androgen receptors. 

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PCOS-Related Hair Loss

Polycystic Ovary Syndrome (PCOS) affects approximately 1 in 10 women of reproductive age, making it one of the most common endocrine disorders today. While often associated with irregular periods, acne, or weight fluctuations, one of the most distressing and overlooked symptoms is hair loss—particularly diffuse thinning around the crown and temples.

Understandinghow and why PCOS triggers hair loss can help women take action toward managing the root causes and supporting healthier, stronger hair regrowth.

The Role of Insulin Resistance in PCOS Hair Loss

PCOS is marked byinsulin resistance—a condition where the body’s cells become less responsive to insulin, prompting the pancreas to produce more of it.

Why This Matters for Hair:

• High insulin levels stimulate the ovaries and adrenal glands to produce even more androgens, including DHT.
• This leads to a vicious cycle: more insulin → more androgens → more follicle miniaturization.
• Insulin resistance also drives inflammation, increases oxidative stress, and disrupts the skin’s microcirculation—factors that compromise scalp health and impair follicle function.

Women with PCOS often have a double-hit effect: elevated DHT from hormonal imbalance, plus worsening inflammation and nutrient delivery issues due to insulin resistance.

How PCOS Triggers Hair Loss: The Androgen Connection

Another key mechanism in PCOS-related hair loss is a disruption in hormonal balance, particularly with elevated androgens—male sex hormones like testosterone and dihydrotestosterone (DHT) that are present in all women in smaller amounts.

Elevated Androgens Shrink Hair Follicles

In women with PCOS, ovarian dysfunction and insulin resistance lead to increased production of androgens. These androgens—especially DHT—bind to receptors in scalp hair follicles, just as they do in androgenetic alopecia.

• DHT attaches to androgen receptors in the hair follicle’s dermal papilla.
• This signals the follicle to shrink, or miniaturize, over time.
• As a result, the hair growth cycle is shortened, and finer, weaker hairs are produced.
• With each cycle, the follicle becomes less productive—eventually leading to noticeable or excessive shedding.

This form of androgen-driven hair loss in PCOS often resembles female pattern hair loss, but can appear earlier and progress more quickly, particularly under chronic hormonal imbalance.

Inflammation, Stress & the HPA Axis

PCOS can also lead to HPA axis dysfunction, meaning the body's central stress system is chronically overstimulated. This results in:

• Elevated cortisol, which suppresses the growth phase (anagen) of the hair cycle.
• Increased inflammation and oxidative stress, which damage follicles.
• A more hostile scalp environment, reducing the follicle's ability to regenerate thick, healthy strands.

Together, these internal imbalances create a perfect storm for thinning hair.

What Makes PCOS Hair Loss Different?

Unlike male-pattern baldness, PCOS hair loss typically presents as:

Diffuse thinning across the crown or wider part line.

• Hair that grows slower, is weaker, and breaks more easily.
• Persistent shedding despite a healthy lifestyle—especially during periods of stress, poor sleep, or unbalanced blood sugar.

Many women also experience increased body hair (hirsutism) while losing hair from the scalp—another sign of androgen dominance.

How to Support Hair Health with PCOS

Treating PCOS-related hair loss requires a multi-pronged approach:

1. Address Insulin Resistance

One of the most effective ways to support hair health with PCOS is by improving insulin sensitivity, as excess insulin can drive androgen production and inflammation.

• Focus on a balanced, low-glycemic diet that helps stabilize blood sugar levels and reduce hormonal swings.
• Incorporate regular physical activity, such as strength training or moderate cardio, to support metabolic health.
• Consider working with a nutritionist or hormone specialist to explore supportive dietary changes and natural supplements. Be sure to speak with an endocrinologist to determine the best course of action for your unique profile.

2. Support Hormonal Balance & Androgen Regulation

• Because elevated androgens like testosterone and DHT contribute to follicle shrinkage and shedding, hormonal balance plays a central role in PCOS-related hair loss.
• Prioritize stress reduction, consistent sleep, and anti-inflammatory foods, all of which help balance the endocrine system.
• Many women also benefit from targeted lifestyle strategies that support hormonal detoxification and adrenal resilience. It’s essential to consult an endocrinologist who can evaluate your full hormonal profile and guide you toward the most effective options for your needs.

3. Support Hair Growth at the Follicle Level

While internal treatments are essential, supporting the hair growth cycle topically with ingredients that block FGF5 and soothe scalp inflammationcan help bridge the gap between active shedding and visible regrowth. 

• In PCOS, hormonal disruption may increase FGF5 activity, shortening the growth cycle and leading to more shedding.
• évolis products feature natural FGF5-inhibiting botanicals clinically studied to support hair density, strand strength, and regrowth—even when internal factors are still being addressed.

Hair recovery with PCOS is possible—it just takes the right combination of science, strategy, and self-care.

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Thyroid Hair Loss (Hypothyroidism & Hashimoto’s)

Thyroid-related hair loss is more common than many realize—especially among women. The thyroid gland plays a crucial role in regulating metabolism, including the metabolic activity of hair follicles. When thyroid hormone levels are out of balance, the hair growth cycle can become disrupted, resulting in shedding, thinning, and poor hair quality.

How the Thyroid Affects Hair Follicles

 

The thyroid produces two key hormones—T3 (triiodothyronine) and T4 (thyroxine)—that directly influence the cellular turnover rate of hair follicles. Hair follicles are among the most metabolically active structures in the body, so they rely heavily on consistent and adequate thyroid hormone levels to function properly.

When levels drop, particularly in hypothyroidism, the hair cycle slows dramatically. This leads to:

• A shortened anagen (growth) phase, so hair doesn’t grow as long or as thick.
• A prolonged telogen (resting) phase, which results in more hairs being shed.
• Weaker hair shafts, often dry, brittle, and more prone to breakage.

Thinning is often diffuse, affecting the entire scalp, but many women notice it more prominently along the part line, hairline, or crown.

Hashimoto’s Disease: The Autoimmune Component

In cases of Hashimoto’s Thyroiditis, an autoimmune condition, the immune system mistakenly targets the thyroid gland as a threat. Over time, this immune attack impairs thyroid function, leading to chronic hypothyroidism.

But the impact doesn't stop there: autoimmunity can also increase systemic inflammation, which in turn affects the scalp microenvironment and surrounding hair follicles. Inflammatory cytokines—messenger proteins released by the immune system—can directly interfere with follicle signaling, disrupt the hair growth cycle, and even contribute to localized hair loss, especially when flare-ups occur.

This dual impact—hormonal deficiency and immune-related inflammation—makes hair loss from Hashimoto’s particularly challenging and often long-term without intervention.

Supportive Strategies for Hair and Scalp Health

While medical guidance from an endocrinologist is essential for diagnosing and managing thyroid disorders, there are several lifestyle-based and product-based ways to support hair health during thyroid-related disruptions:

• Reduce systemic inflammation: Prioritize a nutrient-dense, anti-inflammatory diet rich in omega-3s, antioxidants, and whole foods to support both thyroid and scalp health.
• Support follicle longevity: Look for products that improve scalp circulation, reduce oxidative stress, and extend the anagen (growth) phase of the hair cycle.
• Treat your Thyroid Condition: If you suspect thyroid issues may be contributing to your hair loss, consult an endocrinologist. They can assess your hormone levels and determine if a medical diagnosis, like hypothyroidism or Hashimoto’s, is the underlying cause. Once identified, you can create a comprehensive plan that combines clinical care with at-home hair support for long-term results.
• Block FGF5: FGF5 is a protein that signals hair follicles to exit the growth phase. In conditions like hypothyroidism where hair is already under stress, blocking FGF5 can help extend the growth window and support denser, healthier strands.

Botanical formulas—such as those used in évolis—target FGF5 and incorporate anti-inflammatory ingredients that help protect and revitalize follicles under stress.

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Stress-Induced Hair Loss (Telogen Effluvium)

Stress-induced hair loss is one of the most common, yet frequently underestimated forms of hair shedding in women. Known clinically as telogen effluvium, this condition occurs when physical, emotional, or environmental stress disrupts the natural hair cycle—triggering a greater proportion of hairs to prematurely enter the telogen (resting) phase. The result is widespread shedding, often noticed in handfuls of hair after brushing or showering.

The Role of Cortisol in Hair Cycle Disruption

The body’s central stress hormone, cortisol, plays a key role in the onset of telogen effluvium. Under chronic stress, the hypothalamic-pituitary-adrenal (HPA) axis becomes overstimulated, signaling a prolonged release of cortisol. 

Elevated cortisol levels directly impact the hair cycle by:

• Shortening the anagen (growth) phase, limiting the time hair has to grow and mature.
• Pushes follicles into the telogen phase prematurely, which leads to excessive shedding.
• Suppresses keratin production, weakening the structure of the hair shaft.
• Triggers inflammation and oxidative stress, damaging the follicle environment and reducing blood flow to the scalp.

These changes create a hostile environment for healthy hair growth. While the follicle is not permanently damaged, it becomes less productive and requires time—and support—to return to its full function.

HPA Axis Dysfunction and Chronic Stress

In cases of prolonged stress or adrenal fatigue, the body’s ability to regulate the HPA axis can break down. This dysfunction further elevates cortisol levels, keeps the body in a prolonged state of alert, and disrupts the communication pathways necessary for hair growth regulation.

The consequences can be widespread:

• Diffuse thinning across the scalp, particularly near the temples and part line.
• Brittle, dry strands that are more prone to breakage.
• Slow regrowth, as the body prioritizes survival over non-essential functions like hair production.

Women who are under chronic emotional stress, recovering from illness, experiencing hormonal fluctuations, or going through traumatic events are at increased risk for telogen effluvium.

What Makes Stress-Related Hair Loss Unique?

Unlike genetic or hormonal hair loss, stress-related shedding is typically temporary—though it can last for several months. Many women find that hair loss continues even after the original stressor has resolved, due to the delayed impact of cortisol on the hair cycle.

Stress-related hair loss often presents with:

• Sudden onset of excessive shedding, especially 2–3 months after a stressful event.
• Hair that appears limp, dull, or less dense.
• A frustrating mismatch between overall health improvements and continued shedding.

The key is not only resolving the stressor—but also restoring the conditions that allow the hair cycle to return to balance.

How to Support Hair Health Under Stress

Managing telogen effluvium requires a multi-faceted approach focused on both internal stress management and external follicle support.

1. Reduce Cortisol & Support the Nervous System

• Implement relaxation techniques like meditation, deep breathing, or yoga.
• Ensure consistent, quality sleep to regulate the HPA axis.
• Consider natural adaptogens (such as ashwagandha or rhodiola) under the guidance of a healthcare provider.

2. Support Scalp Health & Hair Regrowth

• Adopt a nutrient-rich, anti-inflammatory diet high in antioxidants and omega-3s.
• Avoid harsh chemical treatments and excessive heat styling that can exacerbate follicle stress.

<4. Target FGF5 for Faster Recovery

Stress has been shown to increase levels of FGF5, a protein that signals hair follicles to exit the growth phase. Blocking FGF5 can help extend the anagen phase and encourage denser regrowth.

• évolis formulations feature natural, clinically studied FGF5 inhibitors and scalp-boosting botanicals that help protect follicles during recovery and promote longer, stronger strands.

Hair recovery after chronic stress is possible—and incredibly common—but it requires intentional support. By regulating cortisol, restoring balance to the hair cycle, and nurturing the follicle from the outside in, women can take control of stress-related shedding and begin seeing lasting improvement.

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Autoimmune Hair Loss in Women (Alopecia Areata & Lupus Hair Loss)

Hair loss caused by autoimmune disorders presents unique challenges for women. In conditions like alopecia areata and lupus erythematosus, the immune system mistakenly identifies healthy hair follicles as threats and attacks them, leading to varying degrees of hair thinning, patchiness, or baldness.

How Autoimmune Conditions Trigger Hair Loss

In autoimmune hair loss, cytokines—the immune system’s signaling proteins—play a major role in follicle dysfunction. 

These cytokines initiate inflammatory cascades that can:

• Disrupt the anagen phase, prematurely halting active hair growth.
• Trigger apoptosis (cell death) in follicular cells.
• Create localized inflammation in the scalp, altering the follicle’s microenvironment.

In alopecia areata, this process typically results in sudden, round patches of complete hair loss. In systemic lupus erythematosus (SLE), inflammation can cause diffuse thinning, often with scarring if the skin is involved.

What Makes Autoimmune Hair Loss Different?

Unlike other types of hair loss, autoimmune-related shedding can be unpredictable and recurrent. Some key characteristics include:

• Patchy bald spots (alopecia areata) that may regrow and recur.
• Scarring hair loss in lupus, particularly if the scalp lesions become inflamed.
• Hair loss that persists despite healthy lifestyle changes or hair-focused treatments.

Autoimmune-related hair loss often occurs alongside other symptoms such as fatigue, joint pain, and skin changes—making a medical diagnosis and systemic treatment essential.

1. Manage Inflammation from Within

• Adopt an anti-inflammatory diet rich in leafy greens, healthy fats, and antioxidant-rich foods.
• Prioritize gut health to support immune regulation and nutrient absorption.

2. Soothe and Protect the Scalp Environment

• Use gentle, non-irritating hair care products free from sulfates, parabens, and synthetic fragrances.
• Look for formulas with natural anti-inflammatory botanicals such as rosemary oil, mangosteen extract, or licorice root.

3. Extend the Growth Phase with FGF5 Blockers

• Autoimmune stressors may increase the expression of FGF5, contributing to shorter hair cycles and slower regrowth.
• évolis formulas use clinically studied botanical extracts that block FGF5 and help preserve the anagen phase, supporting fuller regrowth over time.

Postpartum Hair Loss (Postpartum Alopecia)

Hair loss after childbirth—commonly referred to as postpartum alopecia—is a well-documented yet emotionally distressing condition that affects many new mothers. While not considered permanent hair loss, the sheer amount of shedding and the sudden nature of it can be alarming. This condition typically begins 1 to 4 months after delivery and is the result of rapid hormonal shifts.

The Hormonal Shift After Pregnancy

During pregnancy, estrogen levels surge, prolonging the anagen (growth) phase of the hair cycle and resulting in thicker, fuller hair. This hormonal elevation effectively “freezes” hair in its growing stage, preventing the usual daily shedding.

After childbirth, however, estrogen levels plummet as the body returns to its pre-pregnancy hormonal state. This abrupt decline triggers many hairs to transition at once from the anagen phase into the telogen (resting) phase—leading to telogen effluvium and the sudden shedding of hundreds of hairs at once.

This shedding is part of the body’s natural postpartum recovery, but the visual impact can feel drastic, especially when paired with physical exhaustion, stress, and changing routines.

Hair Loss After Childbirth: What It Looks Like

Most women notice:

• Significant shedding, particularly around the hairline, temples, and part line.
• Thinner ponytails or scalp visibility in areas where hair was once dense.
• Changes in hair texture, making it feel finer or more fragile.

Although this shedding phase can last for several months, hair typically begins to regrow once hormone levels stabilize. In most cases, full regrowth occurs within 6 to 12 months—though the pace and thickness may vary.

Recovery Timeline & Contributing Factors

Several factors can influence the severity and duration of postpartum hair loss:

• Breastfeeding, which can prolong estrogen suppression and nutrient depletion.
• Sleep deprivation<, which increases stress hormones and delays recovery.
• Nutritional gaps from prenatal depletion and the demands of new motherhood.

Women who experience severe stress, illness, or thyroid imbalances postpartum may also see extended shedding. It’s important to consult a healthcare provider if hair loss continues beyond one year or becomes patchy.

Supporting Hair Recovery After Pregnancy

There are several ways to care for your hair and scalp during this period of natural transition:

1. Nourish From Within

• Focus on a balanced, nutrient-rich diet with protein, iron, biotin, zinc, and vitamin D.
• Stay hydrated and consider gentle, breastfeeding-safe supplements with your doctor’s approval.

2. Protect the Scalp & Follicles

• Use non-stripping, sulfate-free shampoos and avoid tight hairstyles that add tension.
• Minimize heat styling and chemical treatments while your hair is in a fragile state.

3. Target FGF5 to Prolong the Growth Phase

• FGF5 is a protein that signals the end of the anagen (growth) phase. Hormonal stress can elevate FGF5 activity, shortening the cycle.
• évolis products contain plant-based FGF5 inhibitors that help extend the growth window and promote stronger, denser regrowth—supporting your body’s natural recovery process.

Hair loss after childbirth is a normal, temporary response to a profound physiological shift. With a bit of patience and proactive care, you can support your hair through the shedding phase and encourage healthy, vibrant regrowth.

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Perimenopause & Menopause Hair Loss

As women enter midlife, hormonal changes—particularly the sharp decline in estrogen and progesterone—can significantly disrupt the hair growth cycle. These hormones are crucial in maintaining the health and longevity of hair follicles. Their loss during perimenopause and menopause can lead to noticeable thinning, slower regrowth, and changes in hair texture.

Menopausal Hair Changes & Estrogen Deficiency Hair Loss

Under normal conditions, estrogen plays a protective role in the hair cycle by extending the anagen (growth) phase and limiting the effects of androgens like dihydrotestosterone (DHT). DHT is known to shrink hair follicles over time—a process called follicle miniaturization. As estrogen declines during menopause, the body’s ability to buffer against DHT is reduced, allowing it to exert a stronger effect on the scalp.

Here’s what happens scientifically:

• Hair follicles become more sensitive to DHT, which binds to receptors in the follicle’s dermal papilla.
• This initiates a miniaturization process, where follicles shrink in size and produce thinner, shorter, weaker strands.
• The anagen phase shortens, reducing the time hair has to grow and mature.
• More hairs enter the telogen (resting) phase prematurely, increasing daily shedding.
• Reduced estrogen also leads to decreased sebum production, leaving hair drier and more prone to breakage.

This combination of shorter growth phases, increased shedding, and brittle texture often results in diffuse thinning—especially around the part line, crown, or temples. Hair may also change in texture, becoming wirier or more fragile over time.

Postmenopause Hair Loss vs. Aging-Related Thinning

While hair thinning due to menopause is primarily driven by hormonal shifts, aging-related hair loss stems from slower cell turnover, nutrient depletion, and reduced scalp circulation.

Key distinctions:

• Menopausal thinning typically accelerates quickly due to estrogen withdrawal.
• Aging-related thinning occurs gradually and may not present the same sudden change in hair texture or volume.
• Both types may coexist, complicating the appearance and progression of hair loss.

Supporting Hair Health During Perimenopause & Menopause

While hormone therapy may be appropriate under medical guidance, a comprehensive approach is key to maintaining healthy hair during this transitional life stage:

1. Nourish Hormone Balance Naturally

• Adopt an anti-inflammatory diet rich in omega-3s, antioxidants, iron, and phytoestrogens (e.g., soy, flaxseed).
• Reduce stress through meditation, yoga, or breathing techniques to support adrenal function and stabilize cortisol.

2. Revitalize the Scalp Environment

• Use hydrating, non-stripping hair care products that protect fragile follicles.
• Avoid hairstyles and treatments that create tension or exacerbate breakage.

3. Target FGF5 to Extend the Growth Phase

• Hormonal decline can increase the activity of FGF5, a protein that signals hair to leave the growth phase.
• évolis formulations use botanical ingredients that inhibit FGF5 and help preserve the anagen phase, supporting regrowth even during hormone fluctuation.

Menopausal hair thinning is driven by real biological changes—but it can be managed. By addressing hormone imbalances, protecting the follicle, and using science-backed topicals, women can maintain stronger, denser hair well beyond midlife.

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Supporting Healthy Hair Growth

Nutrition & Scalp Health

Proper nutrition is foundational to hair wellness. Key nutrients include:

• Biotin, Zinc & Iron: Support keratin production and follicle metabolism.
• Vitamin D & Omega-3s: Reduce inflammation and support follicle health.
• Antioxidants: Protect against oxidative stress that damages follicles.

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Blocking FGF5 for Thicker Hair

Inhibiting FGF5 helps extend the anagen phase, allowing for longer, denser hair growth. évolis products use botanical ingredients clinically shown to block FGF5.

• Longer Growth Phase: Leads to fuller, thicker hair.
• Botanical Technology: évolis blends support follicles without harsh chemicals.

When to See a Specialist

Persistent hair loss may indicate a medical issue. Seek expert guidance if:

• Shedding exceeds 100 hairs/day for several months.
• You notice sudden or patchy hair loss.
• There is a family history of endocrine or autoimmune conditions.

 

At évolis, we understand that hair loss in women is a deeply personal issue, often driven by a complex web of hormonal shifts, stress, immune imbalances, and life changes like menopause. That’s why we’ve created a science-backed, natural ingredient-based system designed to support healthy hair growth from within. Our goal is to provide a safe, effective way to nourish your scalp and follicles—empowering you to heal and thrive alongside the care of your physician. Because every woman deserves to feel confident, supported, and like her best self.